The Role of Septins in the Human Pathogen Candida Albicans

Des Moines, IA US
October 24, 2014


Target Audience

Des Moines University faculty, staff, and students.


  1. Compare and contrast signaling capacity in phenotypes related to in vivo survival and antifungal drug susceptibility between C. albicans and the non-pathogenic fungus Saccharomyces cerevsiae.
  2. Describe the role that septins have in pathogenesis and antifungal drug susceptibility.
  3. Discuss the impact of removal of the 3' untranslated region of a gene on gene expression.


Jill Blankenship, Ph.D. 
Assistant Professor of Biology, University of Nebraska-Omaha 

Dr. Jill Blankenship has spent the last 16 years studying pathogenic fungi. In her graduate research, she studied antifungal drug targets and their roles in pathogenesis and antifungal drug susceptibility in the human pathogensCryptococcus neoformans and Candida albicans in Joe Heitman's lab at Duke University. In her postdoctoral research in labs at Columbia University and Carnegie Mellon University, Dr. Blankenship used forward genetics approaches to identify genes involved in pathogenesis and antifungal drug susceptibility. She has continued this research in her independent position, and has successfully published 16 research papers and 4 reviews on this topic. 

Course summary
Available credit: 
  • 1.00 CE Contact Hour(s)
Course opens: 
Course expires: 
Event starts: 
10/24/2014 - 12:00pm CDT
Event ends: 
10/24/2014 - 1:00pm CDT
Des Moines University
3200 Grand Avenue
Des Moines, IA 50312
United States

Available Credit

  • 1.00 CE Contact Hour(s)


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